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RTK expression and activity levels were found to be higher in persisters than in drug-sensitive cells, and this difference was crucial to the persisters treatment evasion 31,32

This network also helps in the preservation of correct circadian timing, and its phase delay is adjusted to conform to local physiology ( Circadian Rhythm Genes in Breast Cancer From an epidemiological time point, circadian disruption has been associated with breast cancer (Richards and Gumz, 2013

In vitro , collagen-coated drug-loaded scaffolds supported human tenocyte adhesion, preserved TNMD expression, and increased type I collagen deposition after 14 days, indicating maintenance of tenogenic phenotype and antifibrotic potential (Di Marco et al., 2025)